The sturdy and alertType Bs are usually able to resist many of the most severe diseases common to modern life, such as heart disease and cancer. In fact, a Type B who carefully follows the recommended diet can often bypass severe disease and live a long and healthy life. Type Bs are more prone to immune-system disorders such as multiple sclerosis, lupus, and chronic fatigue syndrome. The Type B Diet is balance and wholesome, including a wide variety of foods. For Type Bs, the biggest factors in weight gain are corn, buckwheat, lentils, peanuts and sesame seeds. These foods have different lectin that affect the efficiency of the metabolic process, resulting in fatigue, fluid retention, and hypoglycemia. The gluten lectin in wheat germ and whole wheat products also adds to the problems cause by other metabolism-slowing foods. It is important to leave off chicken for Type Bs. Chicken contains a Blood Type B agglutinating lectin in its muscle tissue, which attack the bloodstream and potentially lead to strokes and immune disorders. Type Bs thrive on deep-ocean fish, but should avoid all shellfish. The shellfish contain lectins that are disruptive to the Type B system.Type B is the only blood type that can fully enjoy a variety of dairy foods. Most nuts and seeds(especially peanuts, sesame seeds and sunflower seeds) are not advised for Type Bs., they contain lectins that interfere with Type B insulin production. Wheat is not tolerated well by most Type Bs. They contain a lectin that reuce insulin efficiency and failure to stimulate fat “burning”. Rye contains a lectin that settles in the vascular system, causing blood disorders and potentially strokes. Corn and buckwheat are major factors in Type B weight gain, they contribute to a sluggish metabolism, insulin irregularity, fluid retention, and fatigue. Eliminate tomatoes completely from Type B diet. It has lectins that irritate the stomach lining. Fruits and vegetables are generally well tolerated and should be taken generously.

Type B Blood Food List

 Type O – The Food Chart

Reference:

• “Eat Right for Your Type”, by Dr.Peter D”Adamo

• Keep expectations for the holiday season manageable. Manage stress by setting realistic goals and being honest about what you can and cannot do – don’t over commit yourself and you will prevent a lot of seasonal anxiety and pressure. Organize your time by making a list and prioritizing the important activities and spread out (time-wise) commitments to lessen stress and increase enjoyment.
• Don’t drink and drive or let others drink and drive. If you must drink, do so conservatively, limit yourself to one or two drinks interspersed with non-alcoholic beverages and food over a period of time.
• Practice fire safety. Most residential fires occur during the winter months, so be careful to never leave fireplaces, space heaters, stoves, or candles unattended. Make sure fire and carbon monoxide alarms have fresh batteries and are in working order.
  
• Holiday trees are grown as a sustainable crop; in fact, 1 acre of Christmas trees produces enough oxygen to support 18 people, and provides habitats for birds and wildlife. Buying an artificial tree isn’t a better choice, since most faux foliage is made from earth-toxic PVC. The very best thing you can do is buy a live tree with a root ball. Live trees can be found at most nurseries and some tree lots. When you’re done with it, plant the tree in your garden. This requires some forethought, particularly in colder climates where you’ll need to break up the ground for planting before it freezes; you’ll also need to keep a live tree moist, and indoors for no longer than a week. If that’s not possible, purchase your cut tree from a tree farm that shuns pesticides and chemicals.
• Prepare food safely. Remember these simple steps: wash hands and surfaces often, avoid cross-contamination, cook foods to proper temperatures, and refrigerate promptly.
• Eat healthy and get moving. Eat plenty of right for your type fruits and vegetables. Limit your portion sizes and avoid foods high in fat and sugar. Be active for at least 2½ hours a week and help kids and teens be active for at least 1 hour a day.
• Some typical holiday plants can be toxic to people and animals when ingested. Keep pets and young children away from poinsettia, mistletoe and holly berries.
• Leave “yesteryear” in the past and look toward the future. Life brings changes. Each season is different and can be enjoyed in its own way. Don’t set yourself up by comparing today with the “good ol’ days.”
• Do something for someone else – volunteer some time to help others or donate to a food pantry. Across America, families are struggling financially and the need for food and small gifts for children is staggering.
• Prevent money problems—don’t create them. Give gifts of time and yourself, or pull names for gift exchanges. Enjoy activities that are free, such as driving around to look at holiday decorations; going window shopping without buying; making a snowperson with children.
• Don’t Forget the Elderly! Younger family members should try to involve their elders in holiday preparations and make them feel an important part of the family during the holiday season.

MOST IMPORTANTLY…

• Give Thanks. Take a moment to appreciate all that you have. Reflect on your favorite people or the pleasures of the day. Celebrate the moment.

No single diet theory can address all aspects of our individuality, and only a fool would claim that soy, red meat, grains, coconut oil or anything else is universally good or universally bad for everyone.

For example, people who are blood type O appear to derive significant benefit from a diet including hormone and antibiotic free meats and poultry. There is a very basic physiologic reason for this: those with type O blood have almost three times the levels of an enzyme in their intestines called ‘intestinal alkaline phosphatase’ (IAP). [1]. This enzyme performs two very important functions in the body. First, IAP splits dietary cholesterol into smaller fragments, allowing for their proper breakdown. Second, IAP enhances the absorption of calcium from the diet. Now you’d think this was cutting-edge, late-breaking news since it is obviously of tremendous interest in these nutrigenomic times. However, the first observations were made over four decades ago.[2]

In addition to these two critical functions, IAP is an important influence on the ability of the digestive tract to heal. Thus in most of our type O patients (44% of the population) we see a marked improvement in their IBS, colitis and Crohn’s disease when they increase their protein and cut back on their carbohydrates. [3]

Blood type B makes considerable amounts of IAP as well, but type A’s make very little. This probably explains why most studies that have looked at heart disease and blood type show a significantly higher rate of problems with blood type A individuals. These folks really should follow a Mediterranean-type diet.

Later studies showed that type A not only secreted almost no alkaline phosphatase in their intestines, but whatever little they did secrete was in and of itself inactivated by the presence of their own A antigen. [4]

Thus, we have here one of the strongest indications for the long term benefit of a low-fat diet in type A, both with regard to the susceptibility to cardiovascular disease, and (although not mentioned here) their additional susceptibility to cancer. Following the type A eating plan, with its emphasis on a healthy fats, low animal protein and the avoidance of foods high in phenylalanine, is the best method to maximize digestive efficiency in type As, lower their level of intestinal dysfunction, and to influence their susceptibility to cardiovascular disease.

Many medical experts consider the influenza virus (cause of the flu) to be the most dangerous virus in the world. Several times in past history, this virus has been responsible for killing huge numbers of people within a 1 to 2 year period. As an example, the Spanish flu (type A(H1N1)) of 1918-19 killed about 500,000 people in the U.S. and at least 20 million people worldwide. In 1957-58, the Asian flu (type A(H2N2)) resulted in 70,000 deaths in the US and in 1968-69, the Hong-Kong flu (type (A(H3N2)) killed 34,000 in the U.S.

What is the Flu?

Let’s pause here and take a moment to get a clearer picture of what the flu really is. Terminology and language can be fickle and non-specific masters, and so the common day-to-day use of the term flu has evolved to often encompass anything from a common cold to a true flu.

The stomach flu is another misleading term, often used to describe a gastrointestinal illness (the stomach flu is usually not even caused by a virus but by other microorganisms). So, the first critical point to understand is that a flu is not a common cold or a stomach infection.

When researchers, or doctors speak of the flu, they are being very specific and mean an infection by the influenza virus. Epidemic influenza is divided into type A and type B. The most common presentation of influenza includes a fever (usually 100-103 degrees F in adults), respiratory symptoms (such as cough, sore throat, runny or stuffy nose), headache, muscle aches, and often extreme fatigue. So, the second key point is that public health officials and doctors mean influenza virus when they use the term flu.

The year-in, year-out flu can be deadly (in an average year, influenza is associated with about 20,000 deaths), especially for the elderly, immuno-compromised, or those who have an existing condition, such as asthma, diabetes or heart disease. Even for those of us who are in generally good health, the flu can still really “take the wind out of our sails”, causing us to feel miserable for several days to a week or two.

Currently there are three main variants of the flu circulating (two types “A” and one type “B”). The type A variants are the “Hong Kong” type A(H3N2) virus and its relatives (responsible for about 400,000 deaths in the United States since 1968 (90% of which are among the elderly), and distant relatives of the “Spanish Flu”, type A(H1N1). The “H” and “N” refer to viral proteins called haemagglutinin (H) and the neuraminidase (N) (more on this in a bit).

Some medical and public health experts believe it is only a matter of time (in fact they think we are overdue) before a new pandemic (worldwide epidemic) of the flu occurs, killing many, many people.

Why has the flu been able to kill such large numbers of people so quickly in the past? 

I am going to oversimplify here, but follow along.

The flu virus is able to mutate or change over time, allowing it to reinfect you year after year. Usually this is a slow and very gradual process (both type A and B influenza virus can change in this manner). As an example, if you were exposed to last year’s flu virus, your immune system would have created a very specific memory of how to effectively deal with the virus. A new exposure to the same virus would not now be a problem. Since the virus changes a slight amount each year, last years immune memory will partially, but not completely protect you from this year’s influenza infection. Think of it in terms of not seeing a friend for a long while…they will obviously look a bit different, so it might take a moment for you to recognize them and remember their name. However, once this moment passes and you remember the name, you now have a clear idea of how to greet them However, every once in a while, the type A flu virus (the type B does not change in this manner) will have a dramatic and abrupt change to either its haemagglutinin (H) and/or neuraminidase (N) proteins. This results in a new strain of the virus, which is not recognized as something your immune system dealt with in the past. It would be as if a new person moved into your town; you have no information in your memory to identify them as your friend and no idea of the name. In the years that the flu virus became a worldwide epidemic and killed into the millions of people, the flu virus changed in this manner.

Blood type and the flu?

Quite a few different researchers have investigated blood type and influenza. The volume of research alone is almost enough to suggest strong blood type connections, but let’s look at the research just to be sure.

After exposure to the influenza virus, an immune process termed seroconversionshould occur. This means that your immune system should be producing antibodies against the influenza virus. Researchers have found that after circulation of influenza A (type (H1N1) and (H3N2)) and influenza B viruses, the immune response (as measured in a rise in antihaemagglutinin antibodies against the virus) differ along blood type lines.

The following generalized immune observations apply:

Blood type A: Overall has a great ability to generate a quick and substantial antibody response against influenza type A(H1N1) and especially A(H3N2). Their antibody response against influenza B is not quite as dramatic.

Blood type AB: Relatively poor ability to generate high antibody levels against any of the influenza viruses.

Blood type B: Reasonable, but not great ability to generate an antibody response against influenza A(H1N1). Slowest (it can take them 3-5 months) and weakest ability to generate antibodies against influenza A(H3N2) of any blood type. Against influenza B virus, blood type B has a significant advantage and responds differently from either blood group A or O. The blood type B immune response happens much earlier and persists longer.

Blood type O: Relatively decent ability to generate antibody response against influenza A(H1N1) and A(H3N2) viruses. Antibody response against influenza B is not as dramatic as blood type B.

Some researchers have hypothesized that one explanation for the typical emergence of the new epidemic strains of influenza in Asia is connected to blood type (and the relatively high proportion of type B blood found in Asia). It seems that blood type B has a genetic predisposition to latent (chronic) persistence of influenza A virus (especially A(H3N2) “Hong Kong” variants). Often, the influenza virus antigen can still be found in healthy type B individuals as much as 5 months after a flu. This means that although they might not have symptoms, they are providing a safe harbor for the virus.

With these differences in immune responses, we would expect to see differences in susceptibility to and severity of influenza infection between the different blood types…and indeed we do. What we find is that the susceptibility to influenza changes based upon your blood type and the properties of the circulating strains of influenza virus.

Looking at influenza A as a whole, the following blood type generalities exist. People with blood type B are going to be much more susceptible to infection during times when new antigenic variants and serotype’s of influenza virus appear. This is actually particularly bad news for B’s and AB’s, since this is the type of influenza A virus change that results in widespread flu pandemics. Blood type O individuals tend to be susceptible to influenza infection at the period of the circulation of virulent strains (so in years when the flu is making people feel really sick, type O will be hit the hardest). Type A’s are the lucky ones when it comes to influenza A; they have a generalized susceptibility to the less virulent strains of influenza A.

Overall, influenza is probably most problematic year to year for Type AB’s. In general, they are more sensitive to infection by both influenza A and B than the other blood types. They are affected by these viruses earlier and more severely than those with the other blood groups (and they need to be extra cautious regarding an abrupt change in the influenza A virus as well) . Blood type B is most severely affected when the influenza A(H3N2) (this is the “Hong Kong” variety and its relatives) is in circulation; has relatively little difficulty with influenza B and has to be very concerned about an abrupt change in the influenza A strains. Type O gets less influenza A(H1N1) and more A(H3N2). Type A blood indirectly offers relative protection against both strains of influenza A.

Will the flu shot protect me?

Does the flu shot protect the blood types differently? Well, what the research shows is that all blood types will have similar seroconversion frequencies to both the live attenuated and killed subunit vaccines after the administration of TWO doses. But after only ONE dose of the live vaccine, blood type A is much more likely than the other blood types to seroconvert. The lesson to be learned here is that blood types B, AB and O really should probably get two doses of the live vaccine for best results (most type A’s can probably get away with just one dose). With the killed subunit vaccine, type O produces the greatest anti-haemagglutinin antibody response. Again two doses generally places the blood types on equal ground.

In addition to the blood type information, remember the following. This year’s flu shot is made from the most common flu viruses in circulation last year. So, in most years, when the virus changes only a tiny bit from last year, the flu shot will offer some protection.

Note: There are many people nevertheless who benefit significantly from the flu shot including elderly, chronically ill, and immuno-compromised individuals. For more information on who should receive this vaccination it is advised that you contact a physician or the department of public health.

Essentially, the effectiveness of the flu shot is always going to be dependent on how closely the vaccine matches the strain of flu virus in current circulation. So, if the virus changes dramatically from last year (as it did in the pandemic years), the flu shot will be of little to no use, because, in essence, it is not providing you with any one who knows this new person in town. So a key point then with regards to the flu shot is that it offers protection in most years, but probably not from a pandemic flu.

Antivirals and the flu

Amantadine and rimantadine are chemically related drugs that interfere with the replication cycle of influenza type A viruses (they are not effective against influenza type B). They both offer a decent degree of protection against infection if taken daily during flu season; however, cost, compliance, and side-effects limit this type of use for most people.

Amantadine and rimantadine are also useful in treatment of the flu; able to reduce the severity and shorten the duration of influenza A if given within the first 48 hours. One huge drawback with these antiviral’s is that they result in Amantadine- and rimantadine-resistant influenza A viruses when they are used for treatment (a very poor long-term strategy resulting essentially in a possible ineffectiveness of these drugs when you might need them the most for a severe or life-threatening flu).

Zanamivir and Neuraminidase Inhibitors.

Zanamivir was the first in a new class of drugs known as selective viral neuraminidase inhibitors. And, if in fact the old saying that “imitation is the most sincere form of flattery” holds true, this type of drug must hold tremendous promise. Pharmaceutical and biotechnology companies (including one of the industry giants—Hoffman La Roche) have quickly jumped on the neuraminidase band-wagon and are now either planning a launch or are in the process of developing their own neuraminidase inhibitors.

The reason Zanamivir is such a promising development is that in humans it not only prevents influenza infection, but also reduces the duration and intensity of the typical symptoms if given within the first 30 hours during an influenza infection. Let’s take a moment here to discuss haemagglutinins and neuraminidase in the context of influenza (remember these are the H and N in the A(H1N1) and A(H3N2) strains).

The influenza virus forms haemagglutinins (essentially protein spikes) which release an enzyme called neuraminidase in order to spread to new cells and propagate the infection. From a biochemistry perspective, neuraminidase is an enzyme that cleaves terminal sialic acid residues from glycoconjugates (Does the term glycoconjugate remind you of anything? It should, because the antigens on your cells like your ABO marker are glycoconjugates). By cleaving off the sialic acid sugar, the virus can escape from infected cells, spread to new cells, and make the mucus you produce in response to an infection less effective (yes, the runny nose and mucus you produce in response to a cold or flu are actually part of your body’s defense strategy).

It was assumed that an ability to inhibit neuraminidase would be a useful medical intervention for treating (and maybe preventing) the flu. So far in the trials on Zanamivir, this assumption appears to be true. The biggest disadvantage with Zanamivir is that it is not well absorbed orally, so must be given by inhalation. Its use might also be limited by its cost. So far, researchers claim that resistance of the virus to the drug has been only rarely observed (but remains a possible area of concern).

Elderberry and the flu

So where does elderberry fit in this portrait of the flu. I have mentioned it was used historically, but does it work? In experiments, elderberry actually does inhibit replication of all strains of human influenza.

In an actual placebo-controlled, double blind study (the scientific gold-standard so to speak) an extract of elderberry fruit has been shown to be effective for treating influenza B. What this research showed was that people using the elderberry extract got better much quicker (more than 70% were better after 2 days and over 90% of people completely resolved the infection within 3 days). In contrast, the people given a placebo often needed as much as 6 days to feel well.

Why does elderberry work? Well, the researchers found two reasons really. People taking the elderberry were able to produce higher anti-haemagglutination titers to influenza B (meaning their immune system essentially performed better and they now have a higher level of recognition should this flu return). And, elderberry inhibits neuraminidase (yes, that is the same neuraminidase that scientists are spending millions of dollars designing drugs against). (Editor’s note: Zanamivir to an extent duplicates this neuraminidase blocking ability of elderberry, but does not appear to have elderberry’s beneficial impact on the immune system).

An important question that has not been answered yet is…will elderberry work as well against influenza A strains? I don’t have a definitive answer for you on this yet, but based upon its method of action, its in vitro ability, and my clinical observations, the answer is probably yes. Our patients taking the elderberry, blueberry, cherry and apple concentrate mixture, seemed to pass easily through this past flu season. The one word of caution I leave you with is that when it comes to daily use of elderberry, more is not always better. Large doses will lead to nausea. If you are trying to avoid a flu a small amount daily might help. I recommend elderberry especially for type B’s and AB’s because of their general susceptibility to the virus. For treatment we use 2 tablespoons 3-4 times daily for adults and less for children depending upon their body weight.

So, the final key point is…next flu season remember your friendly elderberry.

REFERENCES

1. Naikhin AN, Katorgina LG, Tsaritsyna IM, et al. Indicators of collective immunity to influenza depending on the blood group and sex of the population. Vopr Virusol 1989 Jul-Aug;34(4):419-23 [Article in Russian]
2. Aho K, Pyhala R, Visakorpi R. ABO associated genetic determinant in H1N1 influenza. Tissue Antigens 1980 Oct;16(4):310-3
3. Lebiush M, Rannon L, Kark JD. The relationship between epidemic influenza (A(H1N1) and ABO blood group. J Hyg (Lond) 1981 Aug;87(1):139-46
4. Sominina AA, Tsubalova LM, Karpova LS, et al. Genetic predisposition to latent influenza A virus in children with blood type B(III) as a possible cause of new epidemiologic strains in the countries of South-Eastern Asia. Vestn Ross Akad Med Nauk 1994;(9):21-4 [Article in Russian]
5. Fedorova GI, Slepushkin AN, Popova NS, et al. Correlations of the antigenic specificity of human blood with the levels of antihemagglutinins to influenza viruses. Vopr Virusol 1983 Jan-Feb;28(1):54-7 [Article in Russian]
6. Mackenzie JS, Fimmel PJ. The effect of ABO blood groups on the incidence of epidemic influenza and on the response to live attenuated and detergent split influenza virus vaccines. J Hyg (Lond) 1978 Feb;80(1):21-30
7. Mackenzie JS, Wetherall JD, Fimmel PJ, et al. Host factors and susceptibility to influenza A infection: the effect of ABO blood groups and HL-A antigens. Dev Biol Stand 1977 Jun 1-3;39:355-62
8. Frolov VK, Sokhin AA, Sotnik AY, et al. Polymorphism of human blood groups and incidence of influenza A/Hong Kong (H3N2). Acta Virol 1975 Sep;19(5):406-12
9. Karpova LS, Popova TL, Oleinikova EV, et al. Significance of persons with different blood groups in the influenza type A epidemic process. Zh Mikrobiol Epidemiol Immunobiol 1982;(11):86-91 [Article in Russian]
10. Waghorn SL, Goa KL. Zanamivir. Drugs 1998;55:721-25
11. Zakay-Jones Z, Varsano N, Zlotnik M, et al. Inhibition of several strains of influenza virus in vitro and reduction of symptoms by an elderberry extract (Sambucus nigra L.) during an outbreak of influenza B Panama

Serum and cells are main components of blood. It was noted by Karl Landsteiner, a physician at University of Vienna in Austria long time ago that some sera caused the red blood cell to agglutinate. Dr. Karl’s observation leads to the development of the ABO blood group system. In order to understand blood typing, it is essential to define antibody and antigen. Antibodies are known as immunoglobulin, it is found in serum, whose function is to combat invaders by binding themselves to antigen. Antigen is substance found on the surface of red blood cell, which, when introduced into organism that does not have antigen, it leads to production of antibody. The ABO blood group system is the most important blood type system. Classification of human blood can be divided into type A, type B, type AB and type O. These blood types have certain elements that can make them unique from one another. Because of this difference, one doctor had an idea and he started an international craze when he tried to prove that even in dieting, one plan does not fit all. Eventually, that type of diet was born.

Background

Dr. Peter D’Adamo, a neuropathic physician takes his groundbreaking 15 years of research to the public when he wrote his book Eat Right for your Type. It is all about blood type diet.He used customized programs that will work with genetic makeup in order to maximize health and weight loss as well prevents diseases. His book cites the work of glycobiologist and biochemist who have researched blood groups, implying that their research supports blood type theory.

What is Blood Type Diet?

Blood type diet is a nutrition plan based on blood type. Dr. D’Adamo claims that that diet will not only bring weight loss but can also assist in achieving overall good health status. According to the doctor, a chemical reaction occurs between blood and food we eat. This reaction is said to be caused by a factor called Lectins. Lectins are a type of protein found in food has agglutinating properties that can affect the blood. If the food we eat contains lectins that are incompatible with blood type antigen, the lectins target any organ or body system and begin to agglutinate in the said area. Lectins can cause different problems, especially if they are specific to particular blood type. Fortunately, most lectins are not life threatening, as 95% of it is completely absorbed and sloughed off by the body.

What Are The Different Diets For Each Sort?

Dr. D’Adamo added that it is the restoration of natural genetic rhythm. It will work because it follows logical, clear, scientifically researched plan based on individual cellular profile. The following are some samples of food for each blood type as suggested by Dr. D’Adamo.

Type O

Dr. D’Adamo believed that blood group O is the hunter or the earliest human blood group. This is the reason that people with type O blood should eat the kind of diet the earliest human ate. They are also called carnivores. The diet recommends that this group should eat a high protein diet. They are allowed to eat meat, fish, vegetables and fruits. It is also advised to limit the intake of grains and beans. They are discouraged to eat wheat, beans, corns, cabbage, brussels sprouts and cauliflower. It is also useful to eat different kinds of seafood, red meat, liver, spinach and broccoli as it helps in weight loss.

Type O Food List

• Recommended: meat, fish, vegetables and fruits
• Not Recommended: wheat, beans, corns, cabbage, brussels sprouts and cauliflower
• Recommended for weight loss: seafood, red meat, liver, spinach and broccoli

Type A

Dr. D’Adamo believed that blood group A is the cultivator. They are believed to be the second oldest blood group, appearing about 25000-15000 BC, when human settlements appeared as farming developed. They are also called herbivores. This blood type diet allowed them to consume vegetables, seafood, tofu, beans, grains, legumes and fruits. However, they are prohibited to eat meat, dairy products, kidney beans, lima beans and wheat. Foods that are beneficial to weight loss are vegetables, pineapples, vegetable oil and soy foods.

Type A Food List

• Recommended: vegetables, seafood, tofu, beans, grains, legumes and fruits
• Not Recommended: meat, dairy products, kidney beans, lima beans and wheat
• Recommended for weight loss: vegetables, pineapples, vegetable oil and soy foods

Type B

Dr. D’Adamo believed that blood group B is the nomad or sometimes called omnivores. They are believed to emerge between the periods of 15000-25000 BC. They are associated to have strong immune system and digestive system. Dr. D’Adamo concludes that people with this kind of diet should eat a balance diet. Their blood type diet consists of meat, dairy products, beans, grains, legumes, fruits and vegetables. It is discouraged to consume peanuts, corn, sesame seeds, lentil and wheat. Foods that can aid in weight loss in this blood type diet are eggs, greens, liver and tea.

Type B Food List

• Recommended: meat, dairy products, beans, grains, legumes, fruits and vegetables
• Not Recommended: peanuts, corn, sesame seeds, lentil and wheat
• Recommended for weight loss: eggs, greens, liver and tea

Type AB

Dr. D’Adamo believed that blood group AB is the enigma or the most evolved type. In terms of dietary requirements, this blood type group is a mixed between blood type A and blood type B. Their blood type diet consists of meat, seafood, dairy products, beans, legumes, grains, tofu, fruits and vegetables. It is not recommended for their blood type diet to eat red meat, kidney beans, lima beans and corn. Foods that can help in weight loss are seafood, dairy, greens, tofu and pineapple.

Type AB Food List

• Recommended: meat, seafood, dairy products, beans, legumes, grains, tofu, fruits and vegetables
• Not Recommended: red meat, kidney beans, lima beans and corn
• Recommended for weight loss: seafood, dairy, greens, tofu and pineapple

List Of Foods – What To Eat For Your Kind

Gradual increases in dietary calcium intake above the first quintile in a large female cohort are not associated with further reductions in fracture risk or osteoporosis, according to the results of a prospective longitudinal cohort study reported in the May 24 issue of the BMJ.

“It is problematic to make recommendations regarding calcium intake based on the results from clinical trials and previous cohort studies,” write Eva Warensjö, from Uppsala University in Uppsala, Sweden, and colleagues. “Meta-analyses of randomised trials found that supplemental calcium gave modest or no reduction in risk of fracture. Both the habitual dietary intake of calcium and vitamin D status may affect the outcome and are rarely accounted for in the design of calcium supplementation trials.”

The goal of the study was to evaluate associations between long-term dietary calcium intake and the risk for any type of fractures, hip fractures, and osteoporosis. The study cohort consisted of 5022 women who participated in a subcohort of the Swedish Mammography Cohort of 61,433 women in Sweden who were born between 1914 and 1948. This population-based cohort was established in 1987, and participants were followed up for 19 years for primary outcomes of incident fractures of any type and hip fractures, which were identified from registry data.

A secondary outcome in the subcohort was osteoporosis diagnosed by dual energy x-ray absorptiometry. Repeated food frequency questionnaires allowed determination of dietary consumption.

Of 14,738 women (24%) who had a first fracture of any type during follow-up, 3871 (6%) had a first hip fracture. Osteoporosis was diagnosed in 1012 (20%) of the subcohort. For dietary calcium, the risk patterns were nonlinear. In the lowest quintile of calcium intake, the crude rate of a first fracture of any type was 17.2/1000 person-years at risk vs 14.0/1000 person-years at risk in the third quintile, yielding a multivariable adjusted hazard ratio (HR) of 1.18 (95% confidence interval [CI], 1.12 – 1.25). For a first hip fracture, the HR was 1.29 (95% CI, 1.17 – 1.43), and the odds ratio for osteoporosis was 1.47 (95% CI, 1.09 – 2.00).

The fracture rate in the first calcium quintile was more pronounced with a low vitamin D intake. Although the highest quintile of calcium intake did not further lower the risk for fractures of any type, or the risk for osteoporosis, it was associated with a higher rate of hip fracture (HR, 1.19; 95% CI, 1.06 – 1.32).

“Gradual increases in dietary calcium intake above the first quintile in our female population were not associated with further reductions in fracture risk or osteoporosis,” the study authors write.

Limitations of this study include possible residual confounding; limitations inherent in dietary assessment methods; observational design, precluding conclusions regarding causality; and lack of generalizability to other people of different ethnic origins or to men.

“Dietary calcium intakes below approximately 700 mg per day in women were associated with an increased risk of hip fracture, any fracture, and of osteoporosis,” the study authors conclude. “The highest reported calcium intake did not further reduce the risk of fractures of any type, or of osteoporosis, but was associated with a higher rate of hip fracture.”

Reference:

• www.medscape.org
• The Swedish Research Council

Disturbances in mineral and bone metabolism occur early in the course of chronic kidney disease (CKD). Without prevention or treatment these disturbances progress to renal osteodystrophy encompassing varying combinations and degrees of high- and low-turnover bone disorders.

Although a broad body of literature indicates that load-bearing exercise has favorable effects on bone metabolism in the adult and aged skeleton [1, 2], there are no studies on the impact of exercise on bone in chronic or end stage renal disease (ESRD). However, much of what is known regarding the effects of exercise on bone, particularly in osteoporosis, may be helpful in the preservation of bone strength in these populations. This may be especially relevant due to the contribution of age-associated osteoporosis as the average age of CKD and ESRD patients increases.

While high impact activity is especially osteogenic [3], moderate intensity walking results in modest increases in lumbar bone mineral density (BMD) [4], indicating that low-impact activities can have a beneficial effect. This is an important consideration for individuals with kidney disease since they are prone to fatigue and generally have low exercise capacity [5]. Further, renal osteodystrophy carries increased risk for spontaneous tendon rupture, and low impact exercise is a safer choice. In the walking study, BMD increased due to suppression of bone turnover. Though suppression of bone turnover may be a concern in low-turnover disease, a recent report shows that active ESRD patients with adynamic bone disease have greater mineralized bone volume due to minimodeling compared to less active patients [6]. This suggests that bone formation can continue on a limited basis via this process that appears to be enhanced by physical activity.

Studies in humans including hemodialysis patients [7] show a positive correlation between muscle strength and BMD. This is because mechanical loading applied to the bone by muscle is directly responsible for bone formation and remodeling [8]. Although the effects of strength training on BMD have been equivocal in healthy populations [2], strength and muscle mass do increase in response to strength training in ESRD [9], and thus may benefit bone.

While there is no direct information to support beneficial effects of exercise on the bone disorders in kidney disease, existing information is suggestive of such effects. Because the literature suggests that both low-impact weight bearing exercise such as walking and resistance exercise (strength training) are beneficial to bone, patients with kidney disease should be encouraged to find ways to add these types of physical activity to their daily life. Easy suggestions to encourage more walking include; finding a walking buddy to take walks in the neighborhood, walking the dog, joining a local mall walking program, walking up a flight of stairs instead of taking the elevator, and parking the car further from the store. Local community centers often offer low impact exercise classes, and low to moderate intensity resistance exercise classes, as do many gyms. There are two very good illustrated resistance exercise program guides that patients can download from the National Institute of Aging (www.niapublications.org/exercisebook/index.asp) and from Life Options (www.lifeoptions.org ), another organization that helps educate people with CKD. The NKF also has good information about exercise for people with CKD on its Web site, www.kidney.org. Finally, the overall benefit of physical activity in decreasing the risks of comorbidities and preserving physical function cannot be overlooked, and the recommendation and encouragement of physical activity should be a priority for this population; see www.imakenews.com/ckdupdate/e_article000466465.cfm?x=b11,0,w .

Reference:

1. Wolff I, van Croonenborg JJ, Kemper HCG, Kostense PJ, Twisk JWR: The Effect of Exercise Training Programs on Bone Mass: A Meta-analysis of Published Controlled Trials in Pre- and Postmenopausal Women. Osteoporosis International 9:1-12, 1999
2. Wallace BA, Cumming RG: Systematic Review of Randomized Trials of the Effect of Exercise on Bone Mass in Pre- and Postmenopausal Women. Calcified Tissue International 67:10-18, 2000
3. Heinonen A, Oja P, Kannus P, Sievanen H, Haapasalo H, Manttari A, Vuori I: Bone mineral density in female athletes respresenting sports with different loading characteristics. Bone 17:197-203, 1995
4. Yamazaki S, Ichimura S, Iwamoto J, Takeda T, Toyama Y: Effect of walking exercise on bone metabolism in postmenopausal women with osteopenia/osteoporosis. Journal of Bone and Mineral Metabolism 22:500-508, 2004
5. Johansen KL: Physical functioning and exercise capacity in patients on dialysis. Advances in Renal Replacement Therapy 6:141-148, 1999
6. Ubara Y, Tagami T, Nakanishi S, Sawa N, Hoshino J, Suwabe T, Katori H, Takemoto F, Hara S, Takaichi K: Significance of minimodeling in dialysis patients with adynamic bone disease. Kidney International 68:833-839, 2005
7. Spindler A, Paz S, Berman A, Lucero E, Contino N, enalba A, Tirado S, Santana M, Zeballos A: Muscular strength and bone mineral density in haemodialysis patients. Nephrol. Dial. Transplant. 12:128-132, 1997
8. Chamay A, Tschantz P: Mechanical influences in bone remodeling. Journal of Biomechanics 5:173-180, 1972
9. Kouidi E, Albani M, Konstantinos N, Megalopoulos A, Gigis P, Guiba-Tziampiri O, Tourkantonis A, Deligiannis A: The effects of exercise training on muscle atrophy in haemodialysis patients. Nephrology Dialysis Transplantation 13:685-699, 199

Gelband, a former competitive swimmer, says he discovered the Blood Type Diet® about 7 years ago when a client, a British DJ who is famous on the international nightclub circuit, told Gelband that his doctor recommended that he go on the Blood Type Diet®. Gelband decided to give it a try.

The excercise guru Justin Gelband

“I read the book and started looking at how it works,” Gelband said, “I cut out the avoids, limited the neutrals and loaded up on the beneficial foods. Almost immediately, I had more energy, better digestion, I was leaner and I felt better.”

His own experience prompted him to implement the Blood Type Diet® with “the girls” as he calls them, the models and actresses that he regularly trains.

“I tell them to find out their blood type and buy the book and everyone loves it. They feel better, their skin looks better, they have better digestion and their metabolism gets quicker. Many of them are type O and can’t have dairy or grains – when they cut those out, they feel better.”

Recently Gelband was interviewed for an article on the website Yahoo.com, where he mentions that his clients, the Victoria’s Secret models, follow the Blood Type Diet®. “I got a lot of flack for it on their message board,” he says, “but I feel like saying to these people, ‘How do you know that it doesn’t work until you try it?’” Good point!

Gelband says he doesn’t believe in diets – he believes in finding the right foods that work with your body. “The thing that I like about this (Eat Right 4 Your Type®) is that there is no diet, it just says – these are the foods that are good for you and work with your body. It’s teaching you to eat properly.”

Learning to eat properly is something that Gelband is passionate about, he says that Americans eat far too much processed food and thinks that restaurant portion sizes are far too large. “How can they expect you to eat properly when they give you such huge portions? How do you rationalize that?”

He says that even the organic restaurant around the corner from his New York City studio is guilty of this. “I asked the chef, ‘Why do you give people such a huge platter of food?’ I don’t want to eat 1,200 calories in one meal. Just eat smaller portions that include a little fat, some protein and good carbs.”

In both exercise and with food, Gelband echos Dr. D’Adamo’s mantra of individuality, “Everyone is different. What’s good for you is not right for the next person.”

Source:

• Right 4 your type’s January 2011 Newsletter

In relation to that my own story, I’d love to share with you regarding what Dr. Ann Quasarano, my favorite naturopath, talked about the relation between fertility and your blood type. I believe the following explanation are very useful key for those having problem of infertility.

Happy Pregnant Mom

The decision to have a baby is a happy one for most couples. But for some, the road to pregnancy can be a bumpy one. According to the CDC, 10% of the total reproductive age population experiences some type of difficulty in conceiving a baby. In our lifetime, science has made incredible gains in reproductive technology, including in vitro fertilization, which assists couples in their dream of having a child.

One factor that is often overlooked is blood type. Research has shown that some of the problems associated with fertility result from some form of blood type incompatibility; either between the mother and her fetus, or between the mother and the father. Why would this occur? As you probably already know, each blood type has a chemical marker called an antigen. These blood type antigens protect your body against foreign intruders, such as bacteria, viruses, and parasites. When the antigen encounters a harmful foreign intruder, it creates antibodies against it. The same thing that happens with viruses, bacteria, and food, can also take place when trying to conceive – the body responds by producing antibodies to the sperm or fetus and inhibits conception.

Several studies conducted over the past 40 years have concluded that infertility and habitual miscarriage may be the result of antibodies in the woman’s vaginal secretions reacting with the blood type antigens in the man’s sperm. In one of these studies, it was determined that the preponderance of miscarriages were of type A or type B fetuses, caused as a result of incompatibility with type O mothers who produce anti-A or anti-B antibodies.

What the scientists seem to be missing is that these anti-blood type antibodies are often the result of provocations produced by eating the wrong foods for your type. By eating foods that are beneficial and avoiding foods that trigger the antigen-antibody reaction, you create a hospitable internal atmosphere for conception and the fetus to thrive and grow.

Mom and healthy baby

Blood type incompatibility is, obviously, not the only reason couples may experience infertility, nor does the blood type diet magically resolve all fertility problems, but it makes good sense to approach pregnancy in an optimal state of health. Following the diet right for your type can help to put you on the road to good reproductive health – hopefully, the only bump will be the one on your belly!

Source:

• Right For Your Type®, February 2011 Newsletter